Bone & Joint

Researchers at Columbia University in New York have discovered that certain bone cells produce a hormone called osteocalcin, which influences beta-cell proliferation and controls the metabolism of blood sugar (glucose) and fat deposits through previously unknown mechanisms.

Researchers have used cells from human bone marrow to repair damaged pancreatic beta cells in mice. The study offers hopes that these stem cells could be used to treat diabetes in humans. The stem cells also halted damage to the kidneys caused by diabetes.

Elderly nursing home patients with type 2 diabetes mellitus have higher bone mass and lower bone turnover than those without diabetes.

European Society of Cardiology(ESC) and the European Association for the Study of Diabetes EASD) issue aggressive guidelines for coronary heart disease and diabetes.

Type 2 diabetes mellitus is associated with significantly lower bone turnover and higher results on bone ultrasound measurements, even in a largely vitamin-D-deficient, elderly, female nursing home population.

Activation of peroxisome proliferator-activated receptor (PPAR)-gamma by thiazolidinediones (TZDs) results in lower bone mass.

Bone mineral density (BMD) in premenopausal women with type1 diabetes, is 3 percent to 8 percent lower than in women without diabetes, investigators report.

Bone marrow cells that produced insulin adversely affected nerve cells or neurons.

People in their 70s have a more than 60 percent higher risk of fractures if they also have type 2 diabetes, the most common form of the disease, according to new study findings.

Those with diabetes are ore likely to suffer post-operative complications.Flexible