It is very frustrating when we take our time to educate patients on the complications of diabetes and then they flat out ignore us. These patients usually don’t have symptoms that make them feel bad, and so they feel we are overreacting and don’t really listen or make changes. Usually …

In this week's Homerun Slides, the natural history of type 2 diabetes, the eight core defects implicated in the pathogenesis of type 2 diabetes, and the effects of treatment.

In the final installment of this interview with Dr. Don Fetterolf, Diabetes in Control Publisher Steve Freed asks about recommendations for identifying and treating diabetic foot ulcers.

Type 1A diabetes results from a chronic autoimmune destruction of the pancreatic beta cells, probably initiated by exposure of a genetically susceptible individual to some environmental agent(s). This preclinical period is marked by the presence of autoantibodies to pancreatic beta-cell antigens such as insulin, GAD65 (Glutamic Acid Decarboxylase), ICA512 (called also IA-2) or ZnT8 (Zinc Transporter 8), and precedes the onset of hyperglycemia by a few years.

Safety and efficacy, adaptive control algorithm to be tested in U.S. and Europe.

β-cell centric classification of diabetes puts focus on single common denominator of all types.

New pen may improve glycemic control in people with severely insulin-resistant diabetes.

Current black box warning may be overstating the kidney risk.

New nutrition recommendations filled with numerous changes.

Surgery could lower patients' development of future medical issues.